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PEA, a non-endocannabinoid lipid mediator, has been extensively studied and applied in human therapy due to its analgesic and anti-neuroinflammatory properties. Studies have shown that PEA has good safety and tolerability, especially in its micronized form.

Abundant evidence suggests that PEA can improve cognitive impairment by reducing oxidative stress during neuronal activity loss and enhancing neuronal survival. For example, PEA is a key therapeutic agent for improving cognitive function in Alzheimer's disease (AD), retinal inner layer function in glaucoma patients, or alleviating chronic pain in the elderly.

In conclusion, PEA supplementation can promote cognitive function. These cognitive benefits may even extend to improving well-being and promoting brain health.